Ukmedix has learnt of some research that has enabled scientists to understand a mechanism which regulates the human body's ability to process, store and burn of fat. The research is important because it is through understanding why we store extra fat and why we get obese that new ways of beating obesity may be found and made available to the obese public.

In the research genetically engineered mice had their fat regulating mechanism speeded up and this it was noted prevented them for suffering the effects of a high fat diet and over the course of the research it was seen that these mice dealt with the high fat foods more efficiently and therefore stayed slimmer than the mice that were not modified gentically.

The research was published in the magazine Science a repected scientific journal and the accompanying article talked about the ways in which this research could be used to fight obesity and obesity related diseases such a diabetes and heart problems.

Another interesting aspect of this research was that not only were the mice protected against the weight gain but also they were made immune to fatty liver disease that often occurs with people who are obese and have an insulin resistance problem. The scientists form the the Salk Institute for Biological Sciences based in California saw that the human body's mechanism to keep fat stored needed an enzyme known as ACC and this enzymes activity could be slowed using a protein known as TRB3.

The scientists realised that if they kept the TRB3 pathway active and functioning in the mice the storage of fat would slowly disappear. Overall the scientists noted that the mice with the TRB3 pathway fully functioning were between 10% to 20% slimmer than the other mice and despite eating alot of fatty food they didn't put on any weight over the course of the reseacrh programme.